The Educational Foundation is the scholarship and grant portion of AAUW. Nationally, AAUW is one of the largest sources of grants and scholarships for higher education. For AAUW National, the Branch funds Career Development Grants and Community Action Grants with proceeds from the book sale, bridge group and other fundraisers. On a local level, the Branch funds scholarships at Brookdale Community College, Monmouth University and Douglass College. Every year, we also honor the top female senior math student in each of the 22 local high schools. Our investment in the educational future of woman is our major goal.
Local scholarships:
- Audrie
LaTowsky Scholarship - Brookdale
Community College, Lincroft, NJSelected by Brookdale Community College, the recipient is a permanent Monmouth County Resident and preferably a graduate of an Eastern Monmouth County high school within the branch area or a Monmouth County resident student who is at least 25 years old and is continuing her education. Applications are available from January to May in the Brookdale Financial Aid Office. The scholarship is awarded at the close of the spring semester for use starting the following fall.
- Louise Marsen
Scholarship - Brookdale
Community College, Lincroft, NJ
Selected by the college, the recipient is a female student who is a Monmouth County permanent resident, at least 25 years old, and whose studies are in the Humanities.
Applications are available from January to May in the Brookdale Financial Aid Office. The scholarship is awarded at the close of the spring semester for use starting the following fall.
- Esther
Hymer Scholarship - Monmouth
University, West Long Branch, NJ
Selected by the university, the recipient is a junior or senior female resident of Monmouth County with a high GPA majoring in gender studies, political science, international relations or economics with a focus on women and with financial need.
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Dosis de ciprofloxacina intravenosa. Clinica Chimica Acta, 864 : 8 - 14. 17 Arakawa J.S.H. Rabinowitz D.A. Schulz K.M. 1993. Effects of co-administration piperine, quercetin, and a cycloxygenase inhibitor (clopidogrel) in rat model of angina. Am. J. Physiol. 268 : E931 - E938. 18 Schulz K.M. Arakawa J.S. 2002. A cyclical and paroxysmal cardiac hypertrophy pattern after co-administration of aqueous and piperine in rats. Eur. J. Cardiovasc. Prev. Rehabil. 10 : 571 - 576. 19 Chia S.W. Cui D. Huang L.M. Lee H.K. Koo S.H. Wong B.O. 1996. Effects of piperine on cardiac myocytes in healthy rats. Am. J. Physiol. 268 : E934 - E942. 20 Liu P.J. Zhou H. Huang K.Y. 1997. Cyclic adenosine/piperine-induced myocardial myocyte death of rat heart mitochondria. Am. J. Physiol. 276 : E1725 - E1735. 21 Lee is adderall cheaper than vyvanse O.W.J. Schulz K.M. K.S. Chia S.W. Chua P.H. 1999. The piperine-induced cardiac damage pattern in rat: interaction of adenosine and piperine. Cardiovasc. Res. 42 : 567 - 579. 22 Kim H.O. Cui D. Huang L.M. Wong B.O. 2000. A role for cyclic adenosine monophosphate in the cardiac hypertrophy caused by pyrrolidine and piperine co-administration. Eur. J. Cardiovasc. Prev. Rehabil. 9 : 575 - 583. 23 Lohmann V. Cui D. Huang L.M. Wong B.O. 2001. Cyclic adenosine monophosphate enhances the pyrrolidine and piperidine cardiotoxicity in rat heart mitochondria. where is the cheapest place to fill adderall Am. J. Physiol. 269 : E1579 - E1584. 24 Wong B.O. Cui D. Huang L.M. Lee H.K. Schulz K.S. 2001. The pyrrolidine-induced cardiac hypertrophy pattern in rats can be prevented by co-administration of an adenosine monophosphate supplement. Am. J. Physiol. 270 : E1465 - E1473. 25 Sartori G. Luzzati P. Botta F. Di Chiara S. Nicola E. Caffeine and other central nervous system stimulants do not increase cardiac cell death in a rat model of myocardial infarction. Circulation. 106 : 785 - 787. 26 Liu W.-C. Wang Y.-K. Chen P.J. 2001. A mechanism of pyrrolidine-induced cardiac muscle cell death by adenosine analogues. Mol. Pharmacol. 56 : 474 - 482. 27 online pharmacy to buy hcg M. Hirsch S. C. T. F. Wohl G. O. 1997. An adenosine-like substance from red pepper activates nitric oxide synthesis and inhibits cardiac apoptosis in rat aorta. Am. J. Physiol. 271 : E76 - E81. 28 Liu P.J. Zhou H. Huang K.Y. 1997. A model for the pyrrolidine-induced cardiotoxicity study in brain: interaction between adenosine and pyrrolidine. Cell. Biochem. Mol. Biol. 47 : 1 - 5. 29 Zhang X.-L. Wu X.-Y. 1999 a. Activated adenosine in vitro cardiac mitochondria is mediated through cAMP phosphodiesterases. Biochem. Biophys. Res. Commun. 261 : 1383 - 1385. 29 Zhang X.-L. Wu X.-Y. 1999 b. Activated adenosine as a cause of cAMP in rat heart mitochondria. Biochem. Biophys. Res. Commun. 261 : 1386 - 1389. 30 Wu X.-Y. Xu J.-C. 1999 c. Activated adenosine and cAMP accumulation in murine cardiomyocytes through nitric oxide: the role of reactive oxygen species in cell death. Biochem. Pharm. Bull. 26 : 1087 - 1096. 31 Hao S. Zhang X.-L. Wu Z.-Z. Jiang X. 1998. Cell death by activated adenosine in rat mitral valve. Circ. Res. 83 : 1021 - 1022. 32 Zhou Y.
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Coumadin for factor 5 (M+D ) and for FGF23 23 in response to HFD. C, Representative immunoblots for insulin (IR) and fibrinogen (F) from the liver of mice fed HFD or M+D 5 and/or 23. Data are expressed as mean ± SEM. * and **, P < 0.05 vs. M+D 23. We next analyzed whether these changes in liver transcriptome profile were specific to HFD, or whether these changes were present in other tissues. We compared liver transcriptome profile between the high-fat fed mice M+D 5 or 23 diets. These data indicated that, while HFD promoted hepatic inflammation, M+D 23 diet had no effect cheapest place to get adderall filled on liver transcriptome profile. Therefore, we next analyzed liver transcripts in mice fed normal chow or the wild type (WT) diet. These data indicated that M+D 5 diet also did not lead to increased hepatic inflammation. As indicated in Figure 2, HFD promoted liver inflammation and hepatic steatosis, whereas M+D 5 diet did not. Moreover, in the HFD-fed mice, hepatic inflammation was accompanied by a reduction in mRNA expression for FGF23 and insulin, by increased expression for fibrinogen. In the M+D 23 diet-fed mice, hepatic inflammation was accompanied by increased expression for FGF23, insulin, and β-actin. In the mice fed wild type diet, hepatic inflammation was accompanied by increased expression for fibrinogen. The results in Figures 2 and 3 indicate that, in contrast to HFD, M+D 23 diet was associated with a reduction in the hepatic transcriptome profile associated with decreased expression of FGF23, insulin, and β-actin. In addition, the mice fed M+D 23 diet, expression of fibrinogen was increased. Taken together, these data are consistent with the observations reported for HFD and suggest that the HFD-induced hepatic inflammation contributes to the development of steatosis. In contrast to HFD, the M+D 23 diet had no effect on the transcriptome profile in liver. Indeed, the HFD-induced changes in transcriptome profile liver were not associated with hepatic inflammation. These results have implications in regard to the effect of M+D 23 diet in preventing fat gain and weight loss. (D) Mice were fed ad libitum the M+D 3 and 23 diets were sacrificed after two weeks. The mRNA levels of HSP70 (mRNA: 7.2 where is the cheapest place to get adderall μM), NFATc1a 11.6 CD36 (mRNA: 11.7 μM), and FAS 16.1 μM) were measured by real-time PCR. Data are expressed as mean ± SEM. *, P < 0.05 vs. M+D 3. (C) Hepatic inflammation was associated with decreased expression of hepatic mRNA for FAS, CD36, and HSP 70 in the liver of mice fed high-fat diet. were ad libitum the high-fat diet for 7–12 weeks, and the mRNA levels of enzymes were determined by real-time PCR. (B) Effect of M+D 23 diet on liver transcriptome profile. The profile was measured by qRT-PCR over 6 hours after HFD (n = 7; *P < 0.05 vs. high-fat group) or M+D 23 (n = 7; *P Adderal 30 $125.00 $4.17 $112.50 < 0.05 vs. mice fed normal chow diet). (A) Hepatic inflammation was associated with decreased expression of hepatic mRNA for FAS, CD36, and HSP 70 in the liver of mice fed high-fat diet. were ad libitum the high-fat diet for Phentermine extra strength weight loss pills 7–12 weeks and the mRNA levels of enzymes were determined by real time PCR. To determine the effect of M+D 5 or 23 diets on hepatic inflammation in vivo order to evaluate the underlying mechanism, we first analyzed the transcriptome profile in a cohort of mice fed the high-fat diet (HFD) or M+D 3 (M+D ) is there a cheaper alternative to adderall for 6 weeks, and then at 2 6 weeks after HFD feeding was stopped. As shown in Figure 2A, HFD induced hepatic inflammation, while M+D 5 diet did not. Furthermore, in the HFD group, hepatic inflammation was associated with reduced expression of HSP 70 and CD36 in the liver, with decreased expression of FAS in the liver ( Figures 2B and 2C ). In contrast, M+D 23 diet induced elevated expression of HSP 70 and CD36.